Iga nephropathy to eat better?

We all know that the disease is a serious damage to human health, disease, severe cases will lead to the occurrence of uremia, thereby endangering the lives of patients and health. Therefore, for patients with this disease, not only to pay attention to the treatment of diseases, but also to do care work, especially thecare of the diet.

IgA nephropathy to eat what is good? Also need to pay attention to protein intake.Experts said that low protein can effectively reduce the burden on the kidneys of patients.Therefore, the protein intake needs to be calculated based on patient weight, high-qualityanimal protein food.

2, according to the physical condition of the iga nephropathy, a reasonable diet. Firstnecessary to strictly control the intake of salt, under normal circumstances, patients with a daily salt intake of no more than three grams, but also away from pickled or salty foods. Ifthe patient’s situation is more severe edema, the patient should control the water intake.

Iga nephropathy in patients with severe end, the best of bed rest and avoid too many activities. According to experts, to take the supine position, this can increase renal bloodflow, glomerular filtration rate, thereby reducing the symptoms of edema.

4 Note that the same time, but also pay attention to the iga nephropathy patients with skincare. For the purposes of these patients, a quiet, comfortable environment, but also tomaintain the health of the beds and tidy. The clothes are the best soft cotton clothing.

IgA nephropathy ate? Due to the resistance of patients is relatively weak, but also to avoid the occurrence of a variety of infections, the more common are: urinary tractinfection, upper respiratory tract infection.

Long-term injection of insulin will cause which side effects to patients?

For patients with type 1 diabetes must be from the onset of long-term insulin-dependent diabetic patients with long-term insulin-dependent to the patients and cause which side effects, let to a detailed look.

Long-term injection of insulin will cause which side effects to patients?

Insulin injections side effects: skin infection

Serious infection is rare, but at the injection site from the “red dot” is more common, and dirty skin, lax aseptic injection.

Insulin injections side effects: Insulin edema

Diabetes is uncontrolled, high blood sugar can lead to body water loss, loss of sodium, reduced extracellular fluid. When treated with insulin, glucose effective control 4-6 days, vivo Shuinazhuliu, facial and limb edema, usually within a few days self-absorbed. Initial dose of insulin treatment can occur in different parts of edema, but with the continued use of insulin, can often disappear on their own.

Insulin injections side effects of three: the fat pad

Long-term injection of insulin at the same site to stimulate hypertrophy and hyperplasia of the subcutaneous fat to form a fat pad or nodules, such as insulin injections in fat pads parts, will affect its absorption.

Side effects of insulin injections: subcutaneous ecchymosis

Caused by damage when injected subcutaneous capillaries, congestion is not absorbed, not in this injection. General self-absorbed in a week, without special handling.

Insulin injections side effects: refractive errors

Appeared in the initial insulin therapy, due to the rapid decline in blood sugar, causing the eye lens, vitreous changes in osmotic pressure, water spills resulting lens refraction rate, blurred vision, generally 2-4 weeks healing.

Side effects of insulin injections six: insulin antibodies

Long-term use of animal insulin allows the body to produce insulin antibodies, affect the absorption and hypoglycemic effect of insulin. For insulin release test, will enable insulin measured values ​​low cause results not allowed. Switch to human insulin and insulin analogues can reduce the formation of antibodies.

Insulin injections side effects of seven: weight gain

After injection of insulin often caused by abdominal obesity, common in elderly patients with diabetes. Patients with type II diabetes employing the insulin plus metformin antidiabetic drugs to reduce obesity.

Side effects and hazards of long-term injection of insulin: insulin resistance

Common in obese, insulin dosage is too large in patients with type II diabetes, ketoacidosis, daily insulin dosage greater than 200 units for 48 hours or more can be diagnosed with insulin resistance.

Eight of the side effects of injection of insulin: insulin spillover

When the insulin injection is completed needle injection, the needle will be a small amount of insulin outflow, resulting in insulin dosage are not allowed. Novo Pen injection, such cases are more common. The correct way is: lift the skin when injected 45-degree angle to the needle, if a larger amount can be divided into injection, the amount of small can inject. Wait for one minute and then dialing the needle end of the injection does not appear to insulin spillover phenomenon.

Above is a common long-term insulin injections will give patients the side effects caused by the visible hazards or side effects of long-term insulin injections to patients with very severe, traditional Chinese medicine techniques to provide a new way to type 1 diabetes from insulin Chinese medicine treatment, allowing patients with type 1 diabetes and gradually get rid of insulin, away from complications, and lived a normal life.

How to treat cyst on kidney

Renal cysts after initial treatment, if care is prone to recurrence. Renal cysts recurrentfurther damage the kidneys, and pose a greater threat to health. Shijiazhuang kidney disease hospital experts here briefly explain how to avoid kidney cyst recurrence.

In fact, renal cysts is not terrible, because renal cysts is preventable and curable, take tothe occurrence and development of active treatment can delay and prevent uremia. So once the illness, be sure to take a systematic treatment, the correct treatment can effectively alleviate the condition.

First, control of hypertension. Play a decisive role in the protection of renal function.Renal cysts in patients with impaired renal function before there will be blood pressure,we called the renal cyst disease: the emergence of hypertension will accelerate renaldamage, high blood pressure will be on cardiovascular and cerebrovascular damage willrenal cysts associated with cerebral aneurysms rupture caused by stroke and othercomplications, so the rate of deterioration of renal function, to prevent complications is essential to control blood pressure.

Second, to prevent urinary tract infections. More common in women. In the daily diet,personal hygiene and other aspects should take extra care to appropriate exercise,depending on the weather temperature changes at any time to change clothes, to prevent colds and various infections. If problems are found as early as possible consult a doctorto prevent disease progression.

Third, a reasonable diet. Renal cysts in patients with a reasonable diet is very importantto control the deterioration of renal function progress. Low-salt diet every 2 to 3 grams ofedible salt is appropriate, eat potassium, phosphorus, diet to low protein, low-fat diet, eat foods rich in vitamins and plant crude fiber diet, maintain defecate unobstructed. Notethe high fiber, high cellulose food supplement, not a partial eclipse, whole grains, lean meat, fresh vegetables and fruits, cattle, sheep, pigs, eggs, milk, fish and shrimp, etc.-edible. To avoid alcohol, eat spicy, fried foods; avoid sweets; avoid tonic blind, or not free to believe in advertising supplements, so as not to damage the liver or increasethe burden of the liver.

The clinical manifestations of polycystic kidney disease

Polycystic kidney disease is a hereditary disease, and oppression kidney cyst will continue to increase renal blood and oxygen supply decreased, the kidney ischemia and hypoxia, renal function will be impaired, resulting in elevated serum creatinine, whichcaused by the occurrence of renal failure and uremia. Therefore, renal cysts must be treated in time, then the renal cysts in clinical performance? Pain: for most of the first symptom of polycystic kidney disease, was sustained or paroxysmal ranging from fatigue after the increase. Back or flank pain is the most common symptoms of polycystic kidney disease patients. Age and cyst increases the symptoms became clear that women are more common. Acute pain or pain flare often prompted the cysts rupture, stones or blood clots caused by urinary tract obstruction and co-infection. Chronic pain, enlarged kidneys or cysts stretch renal capsule, renal pedicle, the oppression caused by adjacent organs. The huge hepatic cysts can cause under the right rib pain. Hematuria: for polycystic kidney disease is also the first symptom, the emergence of about half of intermittent painless gross hematuria. More than 90% of patients with intracystic bleeding or gross hematuria. Spontaneous, may also occur after strenuous exercise or trauma. The cause of the hematuria cyst rupture of blood vessels, stones, infections or cancer. General hematuria are self-limiting, 2 ~ 7 days to go away. If bleeding continues more than one week, or patients older than 50 years old, need to be excluded may be cancerous. Renal impairment: early renal damage often appears as kidney function decreased concentration. The majority of patients maintain normal renal cyst growth 40 to 60 years; kidney function began to decline, the rate of decline of glomerular filtration rate per year is about 4.4 ~ 5.5ml/min, the development of impaired renal function to ESRD time is about 10 years. Hypertension: is one of the patients with the most common early manifestation. Young ADPKD patients with normal renal function, 50% of blood pressure greater than 140/90mmHg, while almost 100% in ESRD patients with hypertension. High and low blood pressure and kidney size, cyst how much a positive correlation between age increases continue to rise. These clinical manifestations of polycystic kidney disease. The main manifestations of the polycystic kidney disease renal enlargement, increased pressure in the envelope, pulling the blood vessels and nerves in the renal pedicle gradually worsening pain. This series of symptoms of polycystic kidney disease patients in a timely manner to improve, it is likely to cause disease progression, life-threatening. With surgical excision of the cyst, reducing the oppression of the kidney, symptoms of transient disappeared, a temporary drop in blood pressure, but the cyst growth environment persists, renal intrinsic cells or damaged state, the sac of fluid in the continuous secretion of large cystremoval of free growth space for the growth of small cysts, the method of surgical treatment of congenital polycystic kidney disease is only a temporary stalling tactics over time, small cysts or will again grow up. So it is best infiltration therapy of micro-based traditional Chinese medicine, Chinese medicine active substances in the form of skin penetration through the kidney area, targeted positioning to make it act directly on the lesion site, expansion of the wall of blood vessels, accelerate local blood circulation, quickly take away the cyst fluid . The same time, the application of Chinese medicine active ingredients inactivate the cyst epithelial cells, reducing generate more cystic fluid and intracapsular pressure drop. Cyst inside and outside the formation of the pressure difference between the cyst continuous retraction, so effective in preventing cyst increases radically reduce the cyst on the surrounding renal tissue of oppression, injury, to achieve the purpose of truly effective treatment in vivo environment.

How to prevent the incidence of systemic lupus erythematosus?

Systemic lupus erythematosus, although will not let deadly, but systemic lupus erythematosus, if the cure is not timely, the condition involving the various systems of the body tissues and organs, causing multi-organ diseases. Especially involving the kidney will lead to lupus nephritis, and slowly progress into renal failure, uremia stage, seriously affect the patient’s quality of life, can also cause great harm to the patient’s mind and body.

Especially in recent years, the incidence of systemic lupus erythematosus has increased, many people by the distress and pain of the disease so people are more concerned about a problem, so the prevention of systemic lupus erythematosus.

Patients with systemic lupus erythematosus early general fever, joint pain and other symptoms. The absence of other performance, coupled with the check is not complete, often misdiagnosed as rheumatism, rheumatoid and other disease. Condition misdiagnosed easily lead patients to miss the best treatment time, the further development of the condition of the patient, and ultimately bring serious harm to patients.

What measures should be taken for effective prevention of systemic lupus erythematosus?

Life to develop good habits, daily regular schedule early hours. Try to take time out every day, the amount of physical exercise, physical exercise can enhance the patient’s resistance to diseases, and improve their immune system.

2, in patients with systemic lupus erythematosus should also be taken to avoid direct sunlight, especially in the summer sun is very strong, the patient should be minimized to go out, if you must go out, patients must be prepared to wear a hat or take anti-ultraviolet rays of the sun umbrella.

3, regular inspections. Patients should be checked on a regular basis, if the physical discomfort should seek medical consultation immediately to block the disease at an early stage.

4, good self-test work. Daily life should be noted that concerned about their body changes, if the physical symptoms of erythema, petechiae, ulceration, edema, and timely treatment. Also note that if there is fever, fatigue, weight loss, joint pain, headache and other symptoms can not be ignored, but also for treatment as soon as possible.

The daily diet should also pay attention to light-based, do not eat spicy spicy food, less fat meat, animal offal and other foods high in fat, but also smoking cessation temperance. Eat soft food easily digestible food.

How to do Iga nephropathy patients with proteinuria recurrent

Iga immune complex deposition in the glomerular mesangial area, reducing the glomerular filtration rate, impaired kidney function, and IgA nephropathy patients there will be a series of Clinical symptoms, such as proteinuria. Proteinuria easily repeated.

So what causes patients a lot of proteinuria?

A large number of iga immune complex deposition, causing the glomerular inflammatory response occurs, resulting in increased permeability of the glomerular filtration membrane, so the original can not pass the glomerular filtration membrane macromolecular protein substances easily by leakage filtration membrane. If the large amount of proteinuria of less than timely control the inflammatory response intensified secretion, and finally into a series of factors induced renal toxicity, renal toxicity factors will exacerbate the inflammatory response of the glomerular inflammatory response to expand caused by the inflammation of the entire kidney to promote renal tissue fibrosis.

Under normal circumstances, the protein from the glomerular filtrate through the tubular re-absorption, but the severe inflammatory reaction caused by kidney fibrosis, renal tubules by fibrosis in rats, decreased protein reabsorption capacity . Last glomerular filtered proteins increased the range of out-of-glomerular reabsorption, and tubular long-term overload, this overload reaches a certain level, the tubular reabsorption capacity is a serious decline, resulting in more protein large leakage.

So iga nephropathy patients with proteinuria should be how to do it?

To proteinuria treatment iga nephropathy, need to find the root cause of the trigger proteinuria, start from the root to conduct thorough and effective treatment. In order to prevent patients with proteinuria recurring.

Also understand that proteinuria and the glomerular filtration rate decline due to the fibrosis of the kidney, decreased renal tubular reabsorption capacity, so the treatment is a fundamental block kidney fibrosis, glomerular repair damaged and tubular and inherent in the kidney cells and tissues.

Micro-technology to a new set of Chinese medicine Chinese medicine infiltration therapy is innovation on the basis of renal fibrosis, the use of natural drugs, drugs with special processing. Made of traditional Chinese medicine bag, in pairs of acupoints parts of the patient’s blood circulation to the active ingredients of drugs to reach the kidney lesion sites. Block kidney fibrosis, renal intrinsic cells to repair damaged to rebuild the original structure and function of the kidneys, the kidneys to restore the original function.

Iga nephropathy patients to find the right treatment, do not believe that the recipe sort of calumny. Also have confidence in the cure of their illnesses, low self-esteem and optimism in the face of every day life, to have the courage to overcome the disease.

Hypertension caused end stage renal failure

One, is not the control of hypertension induced deterioration of chronic renal failure progress

Has made it clear, hypertension is both the result of various types of kidney disease, is also an important factor for kidney disease progression. In clinical work, persistent and difficult to be controlled or poorly controlled hypertension is a poor indicator of kidney disease I. The blood pressure is an important independent risk factor for end-stage renal failure.

The majority of chronic end stage renal disease (ESRD) occurs in patients with hypertension, high blood pressure continued existence will further promote the formation of renal fibrosis, accelerate the progress of deterioration of renal function.

Systemic hypertension due to vascular contraction, small vascular sclerosis (AS), renal arteriosclerosis to Erzhi kidney damage; glomerular local blood pressure can lead to high perfusion in the local ball, high pressure and high filtration “three high” state , the glomerular endothelial cell damage, start of renal fibrosis, destruction of the nephron, renal damage, prompting renal function deterioration. Its deterioration of the main reasons for the renin – angiotensin – aldosterone system (RAAS). RAAS in maintaining the body’s normal blood pressure and water and electrolyte physiology balance plays an important role. RAAS is an important part of regulating potassium, sodium balance, blood volume and blood pressure. The progression of chronic renal failure, especially patients with hypertension will have the functional imbalance of the RAAS.

Renin is a proteolytic enzyme, primarily by the juxtaglomerular cells to secrete the release of renin into the blood to the liver to produce angiotensinogen (ANG) hydrolysis of the vascular renin I (Ang I), then the pulmonary circulation the role of vascular tight-converting enzyme (ACE) into vascular compactness factors II (Ang II). A major role in Ang Ⅱ: direct contraction of small arteries, inhibition of renin secretion, increased aldosterone secretion, body sodium and water retention due to increased aldosterone secretion.

Second, the formation of Ang II on renal fibrosis:

1 Ang II in proximal tubular acid-base balance regulation:

Ang II receptor corresponding to the proximal tubular epithelial cells (Ang II R), combined with the reabsorption of sodium, chloride, bicarbonate and water and to participate in the regulation of pH balance:

Ang II glomerular hemodynamics:

Ang II to increase the glomerular efferent arteriole and glomerular capillary tension, vasoconstriction, and thus directly affect the renal blood flow and glomerular filtration rate (GFR). Ang Ⅱ in a direct impact on kidney hemodynamics, causing glomerular capillary pressure and directly promote the development of glomerulosclerosis. But in the early stage of hypertension, sound the regulatory function of the glomerular afferent artery, buffer effect, did not increase glomerular pressure, glomerular pressure at this point is not high, of the glomerular has caused the pathological damage.

3 Ang II increased interstitial osmotic pressure and reduce renal medullary blood supply:

The role of Ang Ⅱ vasoconstriction, increased peritubular capillary and proximal tubular pressure, prompting peritubular vasoconstriction to promote aldosterone secretion, resulting in slide increase in sodium retention and enhance the local lack of renal interstitial infiltrating renal papilla blood, thus affecting the blood supply of the renal medulla.

Ang II affect mesangial cells (MC) proliferation and contraction, abnormal secretion:

(1) Ang II involved in the MC mitogen-activated protein kinase signal transduction pathway, affecting the contractile function of the MC, reducing the glomerular filtration area.

(2) Ang II but also through the activation of MC within the cAMP response element binding protein (CREB), and the promotion of MC from the secretion of various growth factors such as TGF-beta, PDGF, bFGF and IL-6 synthesis and secretion of fiber fibronectin (FN) and collagen Ⅰ, to further promote the proliferative hypertrophy, resulting in extracellular matrix (ECM) accumulation.

5, of RAAS activation increased renin dependent on Ang Ⅱ the role, but withered to the MC receptor-mediated TGF-β synthesis, thereby promoting plasminogen activator inhibitor (PAI) and fibronectin ( FN) in the synthesis of collagen Ⅰ, inhibition of plasminogen activator matter (PA)-mediated degradation of ECM, resulting in degradation of functional decline, the ECM accumulation, glomerulosclerosis and renal fibrosis, induced deterioration in renal function development.

6, Ang II can promote intrinsic glomerular cell apoptosis

(1) combined with Ang Ⅱ receptor activation of signal transduction mechanisms, leading to increased cellular calcium influx, intracellular calcium overload and induce apoptosis.

(2) lead to the intrinsic apoptosis in glomerular glomerular pressure.

(3) Ang II may promote increased synthesis of TGF-beta induced apoptosis.

(4) induce production of reactive oxygen free radicals (OFR) to promote its cytotoxic should lead to intrinsic glomerular cell apoptosis.

Blocking the synthesis of Ang Ⅱ may reduce renal fibrosis caused by Ang II to increase the formation of renal injury, stop the progress of kidney damage.

High creatinine in renal cyst

I am Dr. Sherry Wang. Received the patient laboratory report, compared his recent testing report with his last year in September, some important indexes such as: BUN 23.4, Creatinine 522, Phos 1.33, etc. are rising high gradually, now both kidneys are also detected cysts in it, the diagnosis of Chronic Renal Parenchymal disease can be confirmed. According to the laboratory report, the patient kidney disease is already up to renal failure stage III, with mild Anemia and enlarged prostate. For the patient in this condition, we suggested he should take the proper treatment to control the development of the disease, to prevent it to develop into the uremia stage.

For his disease of renal cyst, it is the most common renal abnormality which can appear at any age. But two thirds of the patients are above fifty years old, and it is regarded as aging changes. Renal cyst is the generic terms of cystic lumps in the kidney. As for the treatment of Renal Cyst, the most important is to restrain the enlargement of the cyst. Our hospital adopts combined therapy of Chinese and Western medicine, that is Micro-Chinese Medicine Osmotherapy. It can quickly remove the toxins in the kidney, improve the environment in the kidney issues, repair the damaged kidney tissues and revive the disordered cell membrane. Besides, it can eliminate the accumulation of glomerular matrix, and recover glomerular filtration function.

The most important thing is his high creatinine, for the Serum creatinine, the creatinine is the metabolites of muscle in human body, it is mainly discharged by Glomerulus Filtration. The concentration of Serum Creatinine changes mainly depending on the filtration ability of glomerulus. Creatinine concentration increases mean Glomerulus’ Filtration ability is weakened. Serum Creatinine testing is mainly used to judge the function of kidney. Kidney itself has a strong reserve and compensation ability, only when the glomerulus’ filtration ability is weakened to 1/3 of the normal will the Serum Creatinine increase significantly.

For the Renal Failure patients, their disease condition can be judged from Serum Creatinine testing value and other relative indexes:

No rise in Serum Creatinine indicates compensatory stage of kidney function;

Serum Creatinine exceeding 176.8μmoI/L indicates the stage of renal failure, if the Serum Creatinine exceeds 200μmo/L and the disease goes worse, it would possibly lead to uremia;

Serum Creatinine exceeding 445μmoI/L indicates stage of Uremia.

We are Shijiazhuang Kidney Disease Hospital of China. Our Hospital is a specialized kidney disease hospital in China. For this patient kidney disease, it is a common disease in our hospital. We treat this kind of kidney disease by the method of Micro-Chinese Medicine Osmotherapy, Immunotherapy and Stem Cell Transplant. At present, the Immunotherapy and Stem Cell Transplant are the most advanced medical technology in treating kidney disease. Many patients from different countries came here to receive this series of Chinese traditional medicine treatment and all of them get good results. For receiving this treatment, the patient must come to China and to live in hospital about one month, because one treatment course is 4 months, after one month treatment, the patient illness condition would be greatly improved, their disease is relatively entered the steady condition, so the patient can bring the medicine for home treatment, the doctors will follow up the patient by phone call.

Stem Cell treatment, as a new medical treatment technology, has been gradually applied to clinical. Mesenchymal Stem Cells (MSC), in particular, has become the hot topic of research due to its advantages of wide source, easy separation and no restraint in ethics. At present, kidney disease is recognized as immune mediated inflammation. Traditional treatment including hemodialysis cannot reverse the aggravation of kidney function. The MSC has the potential of differentiating into kidney tissues and cells and take part in the regeneration and repair after the kidney damage. The stem cells can not only differentiate into various renal intrinsic cells including glomerular endothelial cells, mesangial cells, sertoli cells and renal tubular epithelial cells, but also strengthen the ability of differentiating into the above cells by promoting renal secretion. It can also release the inflammatory reaction of the damaged kidneys and plays an important role in repairing the kidneys.

Talking about the Stem Cell treatment, it is to extract the original, undifferentiated cells and have specially cultured about one week to ten days and then transfused into the patient’s body. Transfusing by vein is adopted with tiny vulnus and painless throughout all the treatment course. It is no need blood matching and will no rejective reaction because of its low immunogenicity, so it can well repair the damaged renal cells and recover renal function for the patients.

As for Chronic Renal Failure, we adopt Micro-Chinese Medicine Osmotherapy and Stem Cell treatment. The two treatments can both repair and restore the renal function. Many patients got effective results through the two treatment methods and lots of foreign patients are receiving these treatments. I hope we can help your patient too.

Treatment schedule:

Take the Chinese traditional medicine to treat his diseases according to the

symptoms and the results of the laboratory report.

Using the Characteristic of Umbilical Mesenchymal Stem Cell transplant assisted by Micro-Chinese Medicine Osmotherapy to treat Renal Failure. Carry out one treatment course of Micro-Chinese Medicine Osmotherapy (4 months) and one course of Stem Cell of Umbilical Mesenchymal ( two times), by way of combining of oral taken and external application. The Micro-Chinese Medicine Osmotherapy treatment 3 hours a day (one hour in the morning, and one hour in the afternoon and one hour in the evening), with the aim of remove the blood stasis and the blood toxin, dilate blood vessels, promote the microcirculation of the whole body, and creating a good environment for the Stem Cell Transplant. In this way, the Stem Cell with the assistant of Micro-Chinese Medicine can block renal fibrosis, repair residual nephron, so as to improve the renal function and increase Glomerulus Filtration Rate.

After treatment, all the patients get great change in their every indexes and symptoms in different levels.

How the treatment of diabetic nephropathy

Diabetes patients and their families have made quite a headache, kidney disease on the merger, there are a lot of people will not stand such a blow, or even frustrated, simply do not cure, can long to live on much longer, in fact, this negative thinking is not right , although diabetic nephropathy is a chronic disease, but medicine is so advanced, diabetic nephropathy can be treated.

How treatment of diabetic nephropathy? Because the typical symptoms of diabetic nephropathy, are usually symptomatic treatment should first be strict control of blood sugar, chronic high blood sugar, the main reason is the cause of microvascular disease of diabetic nephropathy. Control of blood glucose by insulin subcutaneous continuous infusion therapy, can effectively control the progression of nephropathy; effective control of hypertension, high blood pressure not only to accelerate the progress of diabetic nephropathy glomerular damage, and aggravate diabetic nephropathy retinal lesions. Effectively control discharge to reduce high blood pressure can make the urine protein allows reduced kidney function to slow down and prolong life; again active treatment of urinary tract infections because the infection may lead to complications, accelerate damage to the kidneys.

How the treatment of diabetic nephropathy? Shijiazhuang kidney disease hospital used in hospitals for the treatment of micro-based medicine penetrate therapy + stem cells to treat diabetic nephropathy, on the one hand by stem cell therapy can maximize repair damaged kidney cells in the lesions, blocking the process of renal fibrosis, lower blood creatinine, to reduce the complications of renal anemia, hypertension, renal edema, protein, occult blood, and other indicators also enable the reduction or even negative, can prolong dialysis time interval, or even get rid of dialysis, at the same time regulate and control the primary disease . Micro-based traditional Chinese medicine penetration therapy, is a Chinese herb in the treatment of diabetic nephropathy, not only to avoid the drawbacks of oral important block the development of renal fibrosis, and fundamentally, for stem cells has been to create a favorable treatment. In the application of micro-based traditional Chinese medicine penetration blocking therapy to insulin resistance, combined with the stem cells to repair treatment for damaged islet cells and islet repair, repair in patients with pancreatic islet function for therapeutic purposes.

What are the clinical manifestations of diabetic nephropathy

Diabetic nephropathy is a factor of threat to human health, and now more and more patients with diabetes, diabetic nephropathy, the number will increase. Diabetic nephropathy What are the clinical manifestations? What are the implications for the human will.

1, proteinuria

Early diabetic nephropathy without clinical proteinuria, only RIA in order to detect microalbuminuria. The diagnostic criteria of diabetic nephropathy, only the performance of clinical diabetic nephropathy proteinuria, proteinuria gradually developed from intermittent to persistent.

Increase in albumin excretion in the urine of diabetic nephropathy, the use of sensitive radioimmunoassay to detect the so-called microalbuminuria. Microalbuminuria, initially as intermittent or after exercise, and changed to persistent proteinuria. In the incidence of type 1 diabetes, the first five years is generally not microalbuminuria, type 2 diabetes with microalbuminuria compared with type 1 diabetes as early as. With the aggravation of renal disease, proteinuria increase was non-selective. Diabetic nephropathy enters a period of clinical diabetic nephropathy and persistent proteinuria (> 0.5 g / day), the course is often> 10 years. Diabetic nephropathy in patients with urinary protein excretion is generally <3.0 g / day, a small number of patients with microscopic hematuria, are a more common type, with proteinuria increased, and the gradual emergence of edema and hypertension.

2, edema

Generally do not have edema in patients with early diabetic nephropathy, and a small number of patients reduced plasma protein, may have mild edema, swelling occurs when the 24-hour urine protein> 3 g. Significant edema, found only in the rapid development of diabetic nephropathy. Some patients due to massive proteinuria caused by nephrotic syndrome, the incidence of 5% to 10%, even 26% has been reported poor prognosis of these patients, survival rarely> 5 years. Edema compared with diabetic nephropathy, the diuretic response is poor, due to, among hypoproteinemia, some water and sodium retention due to diabetic nephropathy than the general nephrotic syndrome.

3, high blood pressure

Hypertension is common in patients with diabetic nephropathy. Severe kidney disease with hypertension, and hypertension can accelerate the progression of diabetic nephropathy and deterioration, so the effective control of hypertension is very important.In type 1 diabetes among kidney disease, the prevalence of hypertension than normal does not increase; more type 2 diabetic patients with hypertension, but proteinuria, high blood pressure ratio also increased; kidney disease integrated sign in patients with hypertension, hypertension mostly moderate, a few severe. Late manifestation of diabetic nephropathy.

4, renal insufficiency

Diabetic nephropathy, once the process is, azotemia, uremia and its ultimate outcome.Advances in diabetic nephropathy speed is very different. Sustainable for many years, some patients with mild proteinuria but normal renal function; some patients with urinary protein, can be rapid development of nephrotic syndrome, renal function gradually deteriorated, culminating in uremia.

5, anemia

Obvious azotemia diabetes patients may have mild to moderate anemia, iron therapy is invalid. Anemia, erythropoietin due to an impediment, may be associated with long-term restricted protein diet, azotemia related.

6, other organ complications

Cardiovascular disease, such as heart failure, myocardial infarction; neuropathy, such as peripheral neuropathy, involving the autonomic neurogenic bladder; almost 100% of the severe diabetic nephropathy diabetic retinopathy.